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Stockwell laboratory in 2012, represents an iron-dependent, non-apoptotic cell death modality distinguished by intracellular iron accumulation and lipid peroxidation, culminating in cell membrane disruption and cellular demise ( Disrupted iron homeostasis constitutes a primary trigger for ferroptosis initiation
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It has also been found that the FB1 has the potential to cause systemic toxicity, including neurotoxicity, hepatotoxicity, nephrotoxicity and mammalian cytotoxicity [220]
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