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Το BPC-157 είναι θραύσμα 15 αμινοξέων από γαστρική προστατευτική πρωτεΐνη ανθρώπου και σχετίζεται στη βιβλιογραφία με μονοπάτια σηματοδότησης μέσω FGF
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However, even though there is a demonstrated role of senile plaques on this excitotoxicity that leads to AD, these events do not always correlate, and soluble Aβ might the one responsible for synaptic loss and the symptomatology of AD [35–40]
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